Watch Videos
Videos: 1 - 20 of 507
Aortic dissection in parasternal long axis TTE
Frequently, the descending aorta can be seen just behind the left atrium in the parasternal long axis view on TTE. In this case, a subtle finding is a serious one that must be detected. The linear echodensity across the aorta is an acute dissection of the aorta.
Runtime: 0m:4s
Frequently, the descending aorta can be seen just behind the left atrium in the parasternal long axis view on TTE. In this case, a subtle finding is a serious one that must be detected. The linear echodensity across the aorta is an acute dissection of the aorta.
Runtime: 0m:4s
McConnell\s Sign: RV dysfunction in pulmonary embolus
This subxiphoid TTE clip shows right ventricular dysfunction of the basal and mid portions while the apex still contracts. This is referred to as McConnell\s sign, the eponymous finding in patients with pulmonary embolus.
Runtime: 0m:1s
This subxiphoid TTE clip shows right ventricular dysfunction of the basal and mid portions while the apex still contracts. This is referred to as McConnell\s sign, the eponymous finding in patients with pulmonary embolus.
Runtime: 0m:1s
pericardial hematoma early after open heart surgery
early after open heart surgery(cabg),in the icu,suddenly:hypotension,sinus tachycardia and suspicion of anemia.urgent TEE was done and shows:large localized pericardial effusion(hematoma,confirmed by surgery),compressing right atrium and right ventricle(tamponade) with multiple fibrin strands inside.
Runtime: 0m:9s
early after open heart surgery(cabg),in the icu,suddenly:hypotension,sinus tachycardia and suspicion of anemia.urgent TEE was done and shows:large localized pericardial effusion(hematoma,confirmed by surgery),compressing right atrium and right ventricle(tamponade) with multiple fibrin strands inside.
Runtime: 0m:9s
Haemodynamic monitoring using echocardiography: a trial 1
This is a trial: haemodynamic measurements/ results by echocardiography in comparison with PA-cath-results. about 75 y old patient with cardiogenic shock by occlusion of RCA. Echocardiographic results: 1. contractility by mv-insufficiency: dp/dt about 320 mmHg/s (this shows a distinct impairment of left ventricle). 2. an approach to LVEDP by E/E`: about 12,7 mmHg 3. approach to CVP alternatively RAP: collapsibility of IVC: one can see, that there is no undulation/ collapsibility of IVC, diameter of IVC > 2 cm: RAP about 15-20 mmHg 4. approach to cardiac output: LVOT-VTI about 23,5 cm, LVOT-area 1,53 cm^2 (radius 0,7 cm) > stroke volume about 36 ml; heart rate: 110/min > cardiac aoutout about 3,9 l/min 5. tricupid valve (TV): Vmax: 3,4 m/sec, PAPs 35 mmHg + CVP; ATC of PV-flow 89 msec, PV-VTI 12,3 cm, one can calculates the PVR with two methods: > a. PVR= TV-flow velocity/ VTI of RVOT x 10 + 0,16; in this case you can calculate: PVR= 3,4 m/s / 0,125 m x 10 + 0,16 = 276 dyn*s*cm^-5 > b. PVR= (PAPm-PCWP)/CO x 79,9; in this case I couldn´t measure the PAPm and PAPd because I couldn´t depict a PV-insufficiency-signal by echocardiography. In comparison: the PA-cath-results: PCWP (LVEDP) 13-14 mmHg, CVP 19 mmHg, CO 4,3-4,5 l/min, PVR 285 dyn*s*cm^-5, SVR 890 dyn*s*cm^-5, cardiac power 0,71 W In my opinion is echocardiographic measurements of a few parameters a good option to approach haemodynamic in patient and to make a fast decision of therapeutical treatment in ER or ICU. I´m lookinf forward to any comment, hints, tipps and critic. That could help me to improve my skills in that kind of echocardiographic technique.
Runtime: 0m:34s
This is a trial: haemodynamic measurements/ results by echocardiography in comparison with PA-cath-results. about 75 y old patient with cardiogenic shock by occlusion of RCA. Echocardiographic results: 1. contractility by mv-insufficiency: dp/dt about 320 mmHg/s (this shows a distinct impairment of left ventricle). 2. an approach to LVEDP by E/E`: about 12,7 mmHg 3. approach to CVP alternatively RAP: collapsibility of IVC: one can see, that there is no undulation/ collapsibility of IVC, diameter of IVC > 2 cm: RAP about 15-20 mmHg 4. approach to cardiac output: LVOT-VTI about 23,5 cm, LVOT-area 1,53 cm^2 (radius 0,7 cm) > stroke volume about 36 ml; heart rate: 110/min > cardiac aoutout about 3,9 l/min 5. tricupid valve (TV): Vmax: 3,4 m/sec, PAPs 35 mmHg + CVP; ATC of PV-flow 89 msec, PV-VTI 12,3 cm, one can calculates the PVR with two methods: > a. PVR= TV-flow velocity/ VTI of RVOT x 10 + 0,16; in this case you can calculate: PVR= 3,4 m/s / 0,125 m x 10 + 0,16 = 276 dyn*s*cm^-5 > b. PVR= (PAPm-PCWP)/CO x 79,9; in this case I couldn´t measure the PAPm and PAPd because I couldn´t depict a PV-insufficiency-signal by echocardiography. In comparison: the PA-cath-results: PCWP (LVEDP) 13-14 mmHg, CVP 19 mmHg, CO 4,3-4,5 l/min, PVR 285 dyn*s*cm^-5, SVR 890 dyn*s*cm^-5, cardiac power 0,71 W In my opinion is echocardiographic measurements of a few parameters a good option to approach haemodynamic in patient and to make a fast decision of therapeutical treatment in ER or ICU. I´m lookinf forward to any comment, hints, tipps and critic. That could help me to improve my skills in that kind of echocardiographic technique.
Runtime: 0m:34s
Accesory mitral valve
Accessory mitral valve tissue is a rare anomaly of embryologic development of the endocardial cushion and may cause substantial and progressive obstruction of the left ventricular outflow tract.
Runtime: 0m:10s
Accessory mitral valve tissue is a rare anomaly of embryologic development of the endocardial cushion and may cause substantial and progressive obstruction of the left ventricular outflow tract.
Runtime: 0m:10s
4 to 1 flutter
This patient is in atrial flutter at about 4:1 conduction, with flutter waves seen on the ECG tracing. Because of the limited conduction, the mitral and tricuspid valves opening several times for each ventricular contraction, which is clearly seen in this apical 4 chamber clip.
Runtime: 0m:2s
This patient is in atrial flutter at about 4:1 conduction, with flutter waves seen on the ECG tracing. Because of the limited conduction, the mitral and tricuspid valves opening several times for each ventricular contraction, which is clearly seen in this apical 4 chamber clip.
Runtime: 0m:2s
Apical Thrombus with and without contrast
In this side-by-side pair of transthoracic apical clips, a thrombus is clearly visualized in the LV apex. The left image is without contrast and the right image is with echo contrast.
Runtime: 0m:4s
In this side-by-side pair of transthoracic apical clips, a thrombus is clearly visualized in the LV apex. The left image is without contrast and the right image is with echo contrast.
Runtime: 0m:4s
subaortic membrane with mild aortic regurgitation
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
Definity microspheres helps to visualize the left ventricle
When a patient has poor acoustic windows, echo contrast agents such as Definity can be administered to opacify the cardiac chambers providing an excellent visualization of the endocardial border.
Runtime: 0m:4s
When a patient has poor acoustic windows, echo contrast agents such as Definity can be administered to opacify the cardiac chambers providing an excellent visualization of the endocardial border.
Runtime: 0m:4s
dissecting hematoma of the interventricular septum with biventricular apical small thrombi
In a case of anteroseptal myocardial infarction,TTE apical view shows dissecting hematoma of the interventricular septum with biventricular apical small thrombi(with mild mitral regurgitation).
Runtime: 0m:19s
In a case of anteroseptal myocardial infarction,TTE apical view shows dissecting hematoma of the interventricular septum with biventricular apical small thrombi(with mild mitral regurgitation).
Runtime: 0m:19s
multiple rhabdomyoma tumor1/2
3 month old girl with tuberous sclerosis:multiple rhabdomyoma
Runtime: 0m:55s
3 month old girl with tuberous sclerosis:multiple rhabdomyoma
Runtime: 0m:55s
coronary-left ventricular fistula
TTE parasternal long,short axis and apical views with color doppler recording,show a diastolic flow originating from a hole in the interventricular septum and directed toward the left ventricle.
Runtime: 0m:59s
TTE parasternal long,short axis and apical views with color doppler recording,show a diastolic flow originating from a hole in the interventricular septum and directed toward the left ventricle.
Runtime: 0m:59s
coarctation of the aorta and RHD:mild mitral stenosis
coarctation of the aorta and RHD:mild mitral stenosis:suprasternal,parasternal and apical views with color and cw doppler recording
Runtime: 0m:29s
coarctation of the aorta and RHD:mild mitral stenosis:suprasternal,parasternal and apical views with color and cw doppler recording
Runtime: 0m:29s
Echo "smoke" on TEE
In this TEE clip prior to an electrophysiology procedure for atrial flutter, spontaneous echo contrast or "smoke" can be seen wisping through the left atrium due to blood pool stagnation from the atrial dysrhythmia.
Runtime: 0m:1s
In this TEE clip prior to an electrophysiology procedure for atrial flutter, spontaneous echo contrast or "smoke" can be seen wisping through the left atrium due to blood pool stagnation from the atrial dysrhythmia.
Runtime: 0m:1s
infected malignant pericardial and pleural effusion
about 60 y old patient with known metastasised lung cancer with condition after radiatio and chemotherapy but with distinct progress under that therapy. The patient was admitted to our ER with severe dyspnoe, tachykardia, hypotension, \"kussmaul\"-sign (inspiratory dilated jugular veins) and peripheral low voltage in ECG. In TTE we could see a pleural and pericardial severe effusion with signs of tampoonade (collapse of RA and RV, undulation of transtricuspidal flow > 45%) and dilated IVC. The results of tapping showed an infection (> 500 leucocytes/µl and malignant cells).
Runtime: 0m:10s
about 60 y old patient with known metastasised lung cancer with condition after radiatio and chemotherapy but with distinct progress under that therapy. The patient was admitted to our ER with severe dyspnoe, tachykardia, hypotension, \"kussmaul\"-sign (inspiratory dilated jugular veins) and peripheral low voltage in ECG. In TTE we could see a pleural and pericardial severe effusion with signs of tampoonade (collapse of RA and RV, undulation of transtricuspidal flow > 45%) and dilated IVC. The results of tapping showed an infection (> 500 leucocytes/µl and malignant cells).
Runtime: 0m:10s
Gigantic Left Atrium
Rheumatic Mitral stenosis with SEVERELY++ dilated LA and Moderate-Severe Mitral Regurgitation. Peak velocity over MV of 2.3m/s and a mean gradient of 6.6mmHg. LA measured 8.8cm in PLAX and had an A4CH area of 161cm².
Runtime: 0m:10s
Rheumatic Mitral stenosis with SEVERELY++ dilated LA and Moderate-Severe Mitral Regurgitation. Peak velocity over MV of 2.3m/s and a mean gradient of 6.6mmHg. LA measured 8.8cm in PLAX and had an A4CH area of 161cm².
Runtime: 0m:10s
severe pulmonary embolism
young pregnant woman (38 WOP) with dyspnoea, tachycardia was admitted from another hospital to our icu. We found the echocardiographic picture of an cute cor pulmonale with signs of decompensation. actually no catecholamines was needed. We started anticoagulation with UFH (PTT > 60 sec) and decided to do the sectio with cardiac-surgery-standby. No lysis because of possibility of ablatio of placenta.
Runtime: 0m:28s
young pregnant woman (38 WOP) with dyspnoea, tachycardia was admitted from another hospital to our icu. We found the echocardiographic picture of an cute cor pulmonale with signs of decompensation. actually no catecholamines was needed. We started anticoagulation with UFH (PTT > 60 sec) and decided to do the sectio with cardiac-surgery-standby. No lysis because of possibility of ablatio of placenta.
Runtime: 0m:28s
HNOCM
about 60 y old patient was admitted to our icu with typical clinical afflictions of ACS. Condition after transaortale septal myectomy in the case of HOCM for years. Actually we saw a distinct LV-hypertrophy with midventricular gradient dependent on heart rate. In LVOT we could found a notable acceleration of flow but NO SAM or midsystolic closure of aortic valve. Under the therapy with volume and beta-blocker (esmolol) we could see a rapidly haemodynamic improvement. DON´T give that patient inotropics in cases of haemodynamic instability! What do you think about? Make it sense to implantate a DDD-pacemaker to reach an asynchronous stimulation of the heart (with asynchronous contraction behavour/ cycle of right and left ventricle) to moderate the ventricular gradients?
Runtime: 0m:10s
about 60 y old patient was admitted to our icu with typical clinical afflictions of ACS. Condition after transaortale septal myectomy in the case of HOCM for years. Actually we saw a distinct LV-hypertrophy with midventricular gradient dependent on heart rate. In LVOT we could found a notable acceleration of flow but NO SAM or midsystolic closure of aortic valve. Under the therapy with volume and beta-blocker (esmolol) we could see a rapidly haemodynamic improvement. DON´T give that patient inotropics in cases of haemodynamic instability! What do you think about? Make it sense to implantate a DDD-pacemaker to reach an asynchronous stimulation of the heart (with asynchronous contraction behavour/ cycle of right and left ventricle) to moderate the ventricular gradients?
Runtime: 0m:10s
common atrium
common atrium : absence of the interatrial septum with common atrioventricular valve and 2 av orifices.apical view.
Runtime: 0m:11s
common atrium : absence of the interatrial septum with common atrioventricular valve and 2 av orifices.apical view.
Runtime: 0m:11s
Copyright (c) 2006-2013 Cardiac Echoes and TEEs | EchoJournal. All rights reserved.
