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mediastinal tumor
mediastinal tumor(non hodgkin lymphoma):TTE parasternal long and short axis views ,show the heart surrounded by the big tumor and pericardial effusion.
Runtime: 0m:19s
mediastinal tumor(non hodgkin lymphoma):TTE parasternal long and short axis views ,show the heart surrounded by the big tumor and pericardial effusion.
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massive mitral valve insufficiency with giant left atrium
about 70 y old patient with condition after mitral valve reconstruction and implantation of capentier-ring (30 mm). Actually the patient was admitted to our hospital with prolonged dyspnoea. in TTE I found a giant atrium caused by a distinct mitral walve insufficiency (radius aof PISA about 1,1 cm, regurgitationflow Q about 450 ml/s, vena contracta about 0,8-0,9 cm, regurgitation in pulmonary veins) and a mild to severe mitral valve stenosis (PGmean about 15-18 mmHg!!!!).
Runtime: 0m:44s
about 70 y old patient with condition after mitral valve reconstruction and implantation of capentier-ring (30 mm). Actually the patient was admitted to our hospital with prolonged dyspnoea. in TTE I found a giant atrium caused by a distinct mitral walve insufficiency (radius aof PISA about 1,1 cm, regurgitationflow Q about 450 ml/s, vena contracta about 0,8-0,9 cm, regurgitation in pulmonary veins) and a mild to severe mitral valve stenosis (PGmean about 15-18 mmHg!!!!).
Runtime: 0m:44s
malignant pericardial and pleural effusion with tamponade
very young patient with dyspnoea, tachycardia, silent heart murmur and positive kussmaul-sign (positive Beck-Trias!!). in clinical history a carcinoma with pulmonary, cerebrale and osseous meastases are known. In TTE I could found a distinct pleural effusion with pulmonary consolidation (after tapping we actually know that it is malignant effusion) and a pericardial effusion with beginning tamponade with decrease of transmitral flow in inspiration and increase of transtricuspidal flow in inspiration.
Runtime: 0m:14s
very young patient with dyspnoea, tachycardia, silent heart murmur and positive kussmaul-sign (positive Beck-Trias!!). in clinical history a carcinoma with pulmonary, cerebrale and osseous meastases are known. In TTE I could found a distinct pleural effusion with pulmonary consolidation (after tapping we actually know that it is malignant effusion) and a pericardial effusion with beginning tamponade with decrease of transmitral flow in inspiration and increase of transtricuspidal flow in inspiration.
Runtime: 0m:14s
subaortic membrane with mild aortic regurgitation
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
DDD-Pacemaker-leads
about 70 y old patient with condition after DDD-pacemaker-implantation because of two neurokardiogenic synkopes caused by sick-sinus-syndrom. We did the TEE because of new atrial fibrillation to look after thrombus in LAA (RAA) before we did the electrical cardioversion. Besides we check the position of the pacemaker leads.
Runtime: 0m:47s
about 70 y old patient with condition after DDD-pacemaker-implantation because of two neurokardiogenic synkopes caused by sick-sinus-syndrom. We did the TEE because of new atrial fibrillation to look after thrombus in LAA (RAA) before we did the electrical cardioversion. Besides we check the position of the pacemaker leads.
Runtime: 0m:47s
acute coronary syndrom
about 75 y old patient with typical chest-pain with irradiation of left shoulder, dyspnoea; high-risk-patient (diabetes mellitus type 2, arterial hypertension, hyperlipoproteinaemia). In ECG I found no specific signs for myocardial ischaemia; in TTE no myocardial movement disorder. Using strain-rate-analysis we found a posterior/inf. and small anteroseptal movement disorder. high-sensitive Troponin was positive. In cardiac catheter we found a two vessel-desease (RCA, LAD) with leading high-grade stenosis of RCA.
Runtime: 0m:22s
about 75 y old patient with typical chest-pain with irradiation of left shoulder, dyspnoea; high-risk-patient (diabetes mellitus type 2, arterial hypertension, hyperlipoproteinaemia). In ECG I found no specific signs for myocardial ischaemia; in TTE no myocardial movement disorder. Using strain-rate-analysis we found a posterior/inf. and small anteroseptal movement disorder. high-sensitive Troponin was positive. In cardiac catheter we found a two vessel-desease (RCA, LAD) with leading high-grade stenosis of RCA.
Runtime: 0m:22s
Mild LV-wall disturbance detected by speckle-pattern
About 45 y old patient with the typical clinical afflictions of acute coronary syndrom. in TTE I couldn´t see a LV-wall-motion-disorder. In Speckle-tracking/-pattern study I found a mild LV-wall-motion-disturbance. Troponin was positive. In coronary study we found a high-grade stenosis of RIVA compatible with the results of speckle-pattern-study. What do you think? Does anybody have any experiences with that kind of stuff? Is it a suitable kind of study?
Runtime: 0m:14s
About 45 y old patient with the typical clinical afflictions of acute coronary syndrom. in TTE I couldn´t see a LV-wall-motion-disorder. In Speckle-tracking/-pattern study I found a mild LV-wall-motion-disturbance. Troponin was positive. In coronary study we found a high-grade stenosis of RIVA compatible with the results of speckle-pattern-study. What do you think? Does anybody have any experiences with that kind of stuff? Is it a suitable kind of study?
Runtime: 0m:14s
pda
30y,f,ll edema,eye puffiness,..nephrotic $,cardiomeg in cxr,echo:p.effusion,accidental pda pt is highly echo genic
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30y,f,ll edema,eye puffiness,..nephrotic $,cardiomeg in cxr,echo:p.effusion,accidental pda pt is highly echo genic
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infective endocarditis on mitral stenosis2
infective endocarditis on mitral stenosis:stuck vegetation between the two leaflets of the mitral valve(with eccentric mitral regurgitation and mild aortic regurgitation)
Runtime: 0m:22s
infective endocarditis on mitral stenosis:stuck vegetation between the two leaflets of the mitral valve(with eccentric mitral regurgitation and mild aortic regurgitation)
Runtime: 0m:22s
Lambl's excresence?
In this zoom view of the aortic valve in parasternal long axis TTE, a small filamentous structure is visible on the ventricular side of the valve. Would you characterize this as a Lambl's excresence or would you even comment on it in a report?
Runtime: 0m:3s
In this zoom view of the aortic valve in parasternal long axis TTE, a small filamentous structure is visible on the ventricular side of the valve. Would you characterize this as a Lambl's excresence or would you even comment on it in a report?
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Prominent crista and lipomatous hypertrophy of the interatrial septum
In this apical 4 chamber TTE clip, the interatrial septum is thickened consistent with lipomatous hypertrophy and there is a rounded density in the posterior right atrium consistent with a prominent crista terminalis.
Runtime: 0m:3s
In this apical 4 chamber TTE clip, the interatrial septum is thickened consistent with lipomatous hypertrophy and there is a rounded density in the posterior right atrium consistent with a prominent crista terminalis.
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post-interventional VSD
about 60 y old patient with condition after ablation of high symptomatic ventricular extrasystole in inferior mid-septale position by EPU 2 weeks ago. The patient have had dyspnoea and orthpnoea for 2 weeks: in TTE I could see the echocardiographic picture of a VSD. To evaluate and measure the Qp/ Qs-quotient via VTI in RVOT and LVOT I did a TEE. You can see the echocardiographic picture of an aneurysm in mid-septale inferior position with perforation. The haemodynamic measurement obtained following information: right-ventricular stroke-volumen about 100 ml; LV-stroke-volume about 40 ml, shunt-volume about 60 ml. Qp/ Qs > 2 We have the suspicion of a peri-/postinterventinal VSD caused by ablation in the course of EPU 2 weeks ago.
Runtime: 0m:32s
about 60 y old patient with condition after ablation of high symptomatic ventricular extrasystole in inferior mid-septale position by EPU 2 weeks ago. The patient have had dyspnoea and orthpnoea for 2 weeks: in TTE I could see the echocardiographic picture of a VSD. To evaluate and measure the Qp/ Qs-quotient via VTI in RVOT and LVOT I did a TEE. You can see the echocardiographic picture of an aneurysm in mid-septale inferior position with perforation. The haemodynamic measurement obtained following information: right-ventricular stroke-volumen about 100 ml; LV-stroke-volume about 40 ml, shunt-volume about 60 ml. Qp/ Qs > 2 We have the suspicion of a peri-/postinterventinal VSD caused by ablation in the course of EPU 2 weeks ago.
Runtime: 0m:32s
Is it an ARVD?
about 22 y old patient with new oedema of lower limb. In TTE we could see an enlarged right ventricle with good RV-function (TAPSE about 23 mm, TASV about 11 cm/sec) despite of a pulmonary hypertension of about 45 mmHg + CVP. There is no SCD in family history, no syncope, no arrhythmics. In ECG we could see an isolated right-ventrcular prolongation (QRS 1-3/QRS 4-6 = 1,39!!). What do you think could this be an ARVD? Do you know some specific echocardiographic signs of this kind of cardiomyopathy?
Runtime: 0m:9s
about 22 y old patient with new oedema of lower limb. In TTE we could see an enlarged right ventricle with good RV-function (TAPSE about 23 mm, TASV about 11 cm/sec) despite of a pulmonary hypertension of about 45 mmHg + CVP. There is no SCD in family history, no syncope, no arrhythmics. In ECG we could see an isolated right-ventrcular prolongation (QRS 1-3/QRS 4-6 = 1,39!!). What do you think could this be an ARVD? Do you know some specific echocardiographic signs of this kind of cardiomyopathy?
Runtime: 0m:9s
pericardial effusion with fibrinous strands
pericardial effusion with fibrinous strands,apical view.etiology:tuberculosis.
Runtime: 0m:18s
pericardial effusion with fibrinous strands,apical view.etiology:tuberculosis.
Runtime: 0m:18s
Tachy-Cardiomyopathy
About 50 y old patient with atrial fibrillation (HR about 150-170/min); the patient told clinical afflictions in the course of time of 3 weeks. In TTE we found a distinct LV-motion disorder and decrease of LV-EF. After exclusion of thrombus in LAA by TEE we did the elektrical cardioversion. After 1 week we saw a distinct improvement of LV-function.
Runtime: 0m:17s
About 50 y old patient with atrial fibrillation (HR about 150-170/min); the patient told clinical afflictions in the course of time of 3 weeks. In TTE we found a distinct LV-motion disorder and decrease of LV-EF. After exclusion of thrombus in LAA by TEE we did the elektrical cardioversion. After 1 week we saw a distinct improvement of LV-function.
Runtime: 0m:17s
myocardial infarction : Lv apical mural thrombus
TTE apical view shows,in a case of anterior myocardial infarction,apical akinesia with LV apical mural thrombus.
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TTE apical view shows,in a case of anterior myocardial infarction,apical akinesia with LV apical mural thrombus.
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myocardial infarction : mobile LV thrombus
TTE multiple views show,in a case of anterior myocardial infarction,apical akinesia with mobile apical LV thrombus.
Runtime: 1m:6s
TTE multiple views show,in a case of anterior myocardial infarction,apical akinesia with mobile apical LV thrombus.
Runtime: 1m:6s
Tako Tsubo cardiomyopathy
about 50 y old patient with severe posttraumatic stress disorder caused by death of near relative. The patient was admitted to out ER/ ICU with distinct and typical clinical affliction of acute coronary syndrome. Troponine I 0,8 (normal till 0,023), CK 210 U/l. In ECG we could see repolarisation disturbance without signs of ST-elevations. In TTE you can see myocardial movent disorder of apical LV-wall like an apical ballooning. No disturbance of RV. No US-B-lines, no pleural effusion, no sign of LVOTO despite the echocardiographic picture of hyperdynamic basal LV-wall-segments. In cardiac catheter no sign of coronary disease; we could see in cardiac catheter the typical movement disorder of Tako-Tsubo-CM as we could see in TTE before. One question to the community: Is there ALWAYS a need for cardiac catheter if we see the typical echocardiographic picture of apical ballooning/ Tako-Tsubo-CM?
Runtime: 0m:11s
about 50 y old patient with severe posttraumatic stress disorder caused by death of near relative. The patient was admitted to out ER/ ICU with distinct and typical clinical affliction of acute coronary syndrome. Troponine I 0,8 (normal till 0,023), CK 210 U/l. In ECG we could see repolarisation disturbance without signs of ST-elevations. In TTE you can see myocardial movent disorder of apical LV-wall like an apical ballooning. No disturbance of RV. No US-B-lines, no pleural effusion, no sign of LVOTO despite the echocardiographic picture of hyperdynamic basal LV-wall-segments. In cardiac catheter no sign of coronary disease; we could see in cardiac catheter the typical movement disorder of Tako-Tsubo-CM as we could see in TTE before. One question to the community: Is there ALWAYS a need for cardiac catheter if we see the typical echocardiographic picture of apical ballooning/ Tako-Tsubo-CM?
Runtime: 0m:11s
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