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Videos: 41 - 60 of 507
tumor like mitral annular calcification
TTE parasternal long axis,apical and subcostal views,show a large,pseudotumoral,mitral annular calcification (incidental finding in a diabetic ,hypertensive, 52 year old male patient with mild mitral regurgitation)
Runtime: 0m:35s
TTE parasternal long axis,apical and subcostal views,show a large,pseudotumoral,mitral annular calcification (incidental finding in a diabetic ,hypertensive, 52 year old male patient with mild mitral regurgitation)
Runtime: 0m:35s
mediastinal tumor
mediastinal tumor(non hodgkin lymphoma):TTE parasternal long and short axis views ,show the heart surrounded by the big tumor and pericardial effusion.
Runtime: 0m:19s
mediastinal tumor(non hodgkin lymphoma):TTE parasternal long and short axis views ,show the heart surrounded by the big tumor and pericardial effusion.
Runtime: 0m:19s
massive mitral valve insufficiency with giant left atrium
about 70 y old patient with condition after mitral valve reconstruction and implantation of capentier-ring (30 mm). Actually the patient was admitted to our hospital with prolonged dyspnoea. in TTE I found a giant atrium caused by a distinct mitral walve insufficiency (radius aof PISA about 1,1 cm, regurgitationflow Q about 450 ml/s, vena contracta about 0,8-0,9 cm, regurgitation in pulmonary veins) and a mild to severe mitral valve stenosis (PGmean about 15-18 mmHg!!!!).
Runtime: 0m:44s
about 70 y old patient with condition after mitral valve reconstruction and implantation of capentier-ring (30 mm). Actually the patient was admitted to our hospital with prolonged dyspnoea. in TTE I found a giant atrium caused by a distinct mitral walve insufficiency (radius aof PISA about 1,1 cm, regurgitationflow Q about 450 ml/s, vena contracta about 0,8-0,9 cm, regurgitation in pulmonary veins) and a mild to severe mitral valve stenosis (PGmean about 15-18 mmHg!!!!).
Runtime: 0m:44s
malignant pericardial and pleural effusion with tamponade
very young patient with dyspnoea, tachycardia, silent heart murmur and positive kussmaul-sign (positive Beck-Trias!!). in clinical history a carcinoma with pulmonary, cerebrale and osseous meastases are known. In TTE I could found a distinct pleural effusion with pulmonary consolidation (after tapping we actually know that it is malignant effusion) and a pericardial effusion with beginning tamponade with decrease of transmitral flow in inspiration and increase of transtricuspidal flow in inspiration.
Runtime: 0m:14s
very young patient with dyspnoea, tachycardia, silent heart murmur and positive kussmaul-sign (positive Beck-Trias!!). in clinical history a carcinoma with pulmonary, cerebrale and osseous meastases are known. In TTE I could found a distinct pleural effusion with pulmonary consolidation (after tapping we actually know that it is malignant effusion) and a pericardial effusion with beginning tamponade with decrease of transmitral flow in inspiration and increase of transtricuspidal flow in inspiration.
Runtime: 0m:14s
subaortic membrane with mild aortic regurgitation
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
previously I have posted a part of this clip.today I upload the complete clip concerning a case of subaortic membrane with subaortic systolic gradient(stenosis) and mild aortic regurgitation.TTE parasternal long axis and apical views.
Runtime: 0m:54s
DDD-Pacemaker-leads
about 70 y old patient with condition after DDD-pacemaker-implantation because of two neurokardiogenic synkopes caused by sick-sinus-syndrom. We did the TEE because of new atrial fibrillation to look after thrombus in LAA (RAA) before we did the electrical cardioversion. Besides we check the position of the pacemaker leads.
Runtime: 0m:47s
about 70 y old patient with condition after DDD-pacemaker-implantation because of two neurokardiogenic synkopes caused by sick-sinus-syndrom. We did the TEE because of new atrial fibrillation to look after thrombus in LAA (RAA) before we did the electrical cardioversion. Besides we check the position of the pacemaker leads.
Runtime: 0m:47s
acute coronary syndrom
about 75 y old patient with typical chest-pain with irradiation of left shoulder, dyspnoea; high-risk-patient (diabetes mellitus type 2, arterial hypertension, hyperlipoproteinaemia). In ECG I found no specific signs for myocardial ischaemia; in TTE no myocardial movement disorder. Using strain-rate-analysis we found a posterior/inf. and small anteroseptal movement disorder. high-sensitive Troponin was positive. In cardiac catheter we found a two vessel-desease (RCA, LAD) with leading high-grade stenosis of RCA.
Runtime: 0m:22s
about 75 y old patient with typical chest-pain with irradiation of left shoulder, dyspnoea; high-risk-patient (diabetes mellitus type 2, arterial hypertension, hyperlipoproteinaemia). In ECG I found no specific signs for myocardial ischaemia; in TTE no myocardial movement disorder. Using strain-rate-analysis we found a posterior/inf. and small anteroseptal movement disorder. high-sensitive Troponin was positive. In cardiac catheter we found a two vessel-desease (RCA, LAD) with leading high-grade stenosis of RCA.
Runtime: 0m:22s
myocardial infarction : Lv apical mural thrombus
TTE apical view shows,in a case of anterior myocardial infarction,apical akinesia with LV apical mural thrombus.
Runtime: 0m:24s
TTE apical view shows,in a case of anterior myocardial infarction,apical akinesia with LV apical mural thrombus.
Runtime: 0m:24s
myocardial infarction : mobile LV thrombus
TTE multiple views show,in a case of anterior myocardial infarction,apical akinesia with mobile apical LV thrombus.
Runtime: 1m:6s
TTE multiple views show,in a case of anterior myocardial infarction,apical akinesia with mobile apical LV thrombus.
Runtime: 1m:6s
Condition after cardiac surgery
about 35 y old patient with condition after mitral-valve reconstruction (Carpentier-ring 36 mm) because of severe high-grad symptomatic mitral-valve insufficiency caused by mitral-valve prolapse and implantation of mechanical aortic prosthesis because of bicuspid aortic valve (SJM double tilting disk-prosthesis 25 mm). You can see a small prolapse of posterior mitral valve but no relevant m itral valve insufficiency, no elevated transprothetic gradient (PGmean 2,7 mmHg). I used tissue-imagening to demonstrate mitral-valve movement.
Runtime: 0m:47s
about 35 y old patient with condition after mitral-valve reconstruction (Carpentier-ring 36 mm) because of severe high-grad symptomatic mitral-valve insufficiency caused by mitral-valve prolapse and implantation of mechanical aortic prosthesis because of bicuspid aortic valve (SJM double tilting disk-prosthesis 25 mm). You can see a small prolapse of posterior mitral valve but no relevant m itral valve insufficiency, no elevated transprothetic gradient (PGmean 2,7 mmHg). I used tissue-imagening to demonstrate mitral-valve movement.
Runtime: 0m:47s
Endocarditis of aortic bioprosthesis Part 2 TEE
... in that vid you can see an floating structure high-suscipious for endocarditic vegetation with distinct elevation of transprothetic gradient. Besides we could found a severe dynamic mitral valve regurgitation with excentric regurgitation jet without signs of mitral valve prolapse.
Runtime: 0m:39s
... in that vid you can see an floating structure high-suscipious for endocarditic vegetation with distinct elevation of transprothetic gradient. Besides we could found a severe dynamic mitral valve regurgitation with excentric regurgitation jet without signs of mitral valve prolapse.
Runtime: 0m:39s
Endocarditis of aortic bioprosthesis Part 1 TTE
about 80 y old patient with cardial decompensation (known severe RV-insufficiency) with distinct peripheral oedema, pleural effusion and clinical afflictions of infection with distinct radidly increased inflammatory values. The patient has a complicated clinical history. The most importatnt thing is condition after implantation of aortic bioprosthesis (SJM 23 mm) and within of 1 year an endocarditis of aortic bioprosthesis (early-endocarditis of prothesis!!!) about 2 years ago. But then the patient was treated like a late-endocarditis of prothesis (treatment like endocarditis of native valve), no result of bacterial tests. Actually in TTE we found some interesting things: distinct elevated transprothetic gradient above the aortic bioprosthesis (PGmax. 90 mmHg, PGmean 50 mmHg, max. velocity 4,8 m/sec), middle till hicghgrade mitral-valve insufficiency with excentric regurgitation jet; and a decompensated right ventricle with severe increased pulmonary pressure (PAPsystol. 85 mmHg, high-grade tricuspid insufficiency, RAP assessed on dilatated IVC without collapsility about 10-15 mmHg > PAPs about 100 mmHg!!!!). Look at the Part 2 (TEE).... Unfortunately I couldn´t convert my pictures of distinct elevated transprotehtic gradients.
Runtime: 0m:10s
about 80 y old patient with cardial decompensation (known severe RV-insufficiency) with distinct peripheral oedema, pleural effusion and clinical afflictions of infection with distinct radidly increased inflammatory values. The patient has a complicated clinical history. The most importatnt thing is condition after implantation of aortic bioprosthesis (SJM 23 mm) and within of 1 year an endocarditis of aortic bioprosthesis (early-endocarditis of prothesis!!!) about 2 years ago. But then the patient was treated like a late-endocarditis of prothesis (treatment like endocarditis of native valve), no result of bacterial tests. Actually in TTE we found some interesting things: distinct elevated transprothetic gradient above the aortic bioprosthesis (PGmax. 90 mmHg, PGmean 50 mmHg, max. velocity 4,8 m/sec), middle till hicghgrade mitral-valve insufficiency with excentric regurgitation jet; and a decompensated right ventricle with severe increased pulmonary pressure (PAPsystol. 85 mmHg, high-grade tricuspid insufficiency, RAP assessed on dilatated IVC without collapsility about 10-15 mmHg > PAPs about 100 mmHg!!!!). Look at the Part 2 (TEE).... Unfortunately I couldn´t convert my pictures of distinct elevated transprotehtic gradients.
Runtime: 0m:10s
paravalvular abscess of aortic bioprosthesis
about 70 y old patient with condition after cardiac surgery 2 years ago with implantation of aortic bioprothesis because of severe aortic endocarditis with distinct paravalvular abscess. Actually the patient was admitted to our ER/ hospital with chills, fever and other clinical afflications of severe infection. In TTE we couldn´t found any signs of endocarditis and no elevated transprothetic Gradient of aortic bioprosthesis (SJM 25 mm). In TEE you can see a typical paraprothetic abscess of aortic bioprosthesis. Besides we found a mild till moderate mitral valve insufficiency and a sweeping atrial pacemaker-lead into the right ventricle (condition after implantation of DDDR-pacemaker because of intermittend high-grade AV-block and chronotropic incompetency). Pacemaker leads are definitely free of endocarditic vegetations.
Runtime: 0m:26s
about 70 y old patient with condition after cardiac surgery 2 years ago with implantation of aortic bioprothesis because of severe aortic endocarditis with distinct paravalvular abscess. Actually the patient was admitted to our ER/ hospital with chills, fever and other clinical afflications of severe infection. In TTE we couldn´t found any signs of endocarditis and no elevated transprothetic Gradient of aortic bioprosthesis (SJM 25 mm). In TEE you can see a typical paraprothetic abscess of aortic bioprosthesis. Besides we found a mild till moderate mitral valve insufficiency and a sweeping atrial pacemaker-lead into the right ventricle (condition after implantation of DDDR-pacemaker because of intermittend high-grade AV-block and chronotropic incompetency). Pacemaker leads are definitely free of endocarditic vegetations.
Runtime: 0m:26s
Type A aortic dissection with bad image on TTE 2
type A aortic dissection with bad image on tte
Runtime: 0m:30s
type A aortic dissection with bad image on tte
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Type A aortic dissection with bad image on TTE 1
when we suspect aortic dissection we have to do a quick transthoracic echo.even when we have bad images on tte,we always get some signs (dilatation of the aorta,suspicion of tear..)which allow to continue explorations:TEE,ct scan..to make the diagnosis.
Runtime: 0m:30s
when we suspect aortic dissection we have to do a quick transthoracic echo.even when we have bad images on tte,we always get some signs (dilatation of the aorta,suspicion of tear..)which allow to continue explorations:TEE,ct scan..to make the diagnosis.
Runtime: 0m:30s
LVOTO caused by downwards tilted mitral-valve bioprosthesis Part 2
...Part 2 of that- in my opinion - very interesting case. We send that patient to cardia surgery with documetated current increased LVOTO-gradient.
Runtime: 1m:19s
...Part 2 of that- in my opinion - very interesting case. We send that patient to cardia surgery with documetated current increased LVOTO-gradient.
Runtime: 1m:19s
LVOTO caused by downwards tilted mitral-valve-bioprosthesis Part 1
about 30 y old patient with condition after implantation of bioprosthesis in mitral-valve- and aortic-valve-position in africa (NO documents available; indication isn´t known: mitral-valve- and aortic stenosis caused by rheumatic fever?? The patient told that the cardiac valves were very stiff.). The patient was admitted to our ER with recurrent subfebrile fever and dyspnoea, In TTE we found a very interesting result: the mitral-valve bioprosthesis seemed to be downwards tilted into the LVOT with obstruction of LVOT (PG mean 45 mmHg!!). The aortic valve was presented tenuous without pathological gradient but a little bit to small in its diameter.
Runtime: 0m:10s
about 30 y old patient with condition after implantation of bioprosthesis in mitral-valve- and aortic-valve-position in africa (NO documents available; indication isn´t known: mitral-valve- and aortic stenosis caused by rheumatic fever?? The patient told that the cardiac valves were very stiff.). The patient was admitted to our ER with recurrent subfebrile fever and dyspnoea, In TTE we found a very interesting result: the mitral-valve bioprosthesis seemed to be downwards tilted into the LVOT with obstruction of LVOT (PG mean 45 mmHg!!). The aortic valve was presented tenuous without pathological gradient but a little bit to small in its diameter.
Runtime: 0m:10s
acute pulmonary embolism
about 90 y old patient with sudden drop down without dyspnoea but hypotension, tachycardia. In TTE I could visualize that echocardiographic picture of RV-ballooning with D-sign of LV. New high grade tricuspid valve insufficiency (PAPs ca. 40 mmHg + CVP) in comparison with prior echocardiographic results. Unfortunately the patient had also a fracture of the right leg with bleeding complication caused by the drop.
Runtime: 0m:9s
about 90 y old patient with sudden drop down without dyspnoea but hypotension, tachycardia. In TTE I could visualize that echocardiographic picture of RV-ballooning with D-sign of LV. New high grade tricuspid valve insufficiency (PAPs ca. 40 mmHg + CVP) in comparison with prior echocardiographic results. Unfortunately the patient had also a fracture of the right leg with bleeding complication caused by the drop.
Runtime: 0m:9s
Another HOCM Part 2
here the TEE of further patient with severe HOCM with SAM of AMS and following high grade mitral valve insufficiency and fluttering aortic valve respectively mesosystolic closure of aortiv valve (unfortunately I still can´t convert my M-Mode-pictures of SAM and mesosystolic closure of aortic valve).
Runtime: 0m:30s
here the TEE of further patient with severe HOCM with SAM of AMS and following high grade mitral valve insufficiency and fluttering aortic valve respectively mesosystolic closure of aortiv valve (unfortunately I still can´t convert my M-Mode-pictures of SAM and mesosystolic closure of aortic valve).
Runtime: 0m:30s
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