Hi everybody. My name is Pablo, Critical care physician from Argentina. At first look, except for moderator band that seems appear in RV... looks like the transducer are inverted and right structures in reality are left. If not, The IVS is paradoxical. So, if this are right structures, a significant tricuspid regurgitation (annulus dilatation) must be present and sPAP (in sum with IVC measurement) can be estimated. I think pressure and volume overload of RV is present and we must look if a interventricular and/or interauricular communication appear.(volume overload) If not, like Diego says, the clinical history is very important thinking in a lot of causes of PH. Best, Pablo
hello everybody
i think we should consider congenital anomalies
like PAPVC
WHAT DO U THINK?
Hi everybody. My name is Pablo, Critical care physician from
Argentina. At first look, except for moderator band that seems appear
in RV... looks like the transducer are inverted and right structures
in reality are left.
If not, The IVS is paradoxical.
So, if
this are right structures, a significant tricuspid regurgitation
(annulus dilatation) must be present and sPAP (in sum with IVC
measurement) can be estimated.
I think pressure and volume
overload of RV is present and we must look if a interventricular
and/or interauricular communication appear.(volume overload)
If
not, like Diego says, the clinical history is very important thinking
in a lot of causes of PH.
Best,
Pablo
Could you please provide any additional infos about clinical history
and in case Pulmonary pressures?
Thanks for sharing tho'