not a cardiomyopaty ,mitral P2 prolapse probably barlow

how can you say it's cardiomyopathy? from this clip alone I would have
said organic MR (P2 prolapse?) with LV enlargement and moderate LV
dysfunction....

incredible

DILATED CARDIOMYOPATHY...SHOWING 2 CHAMBER ENLARGEMENT LA,LV PROVABLY
4 CHAMBER DILATION..CRITICAL IMPAIRED SYSTOLIC
FUNTION...ETIOLOGIES...IDIOPATIC,TOXINS(ALCOHOL,MEDICATIONS,COBALT,SNA
KE BITES),METABOLIC(THIAMINE
DEFICIENCY,ACROMEGALY,PERIPARTUM)..INFECTIONS(CHAGA"S
DISEASE)....PHYSIOLOGY..1-IMPAIRED LV CONTRACTILITY.2-REDUCE
CO(STARLING LAW NOT APPLY(THE SAME AMOUNT OF BLOOD ENTER THE
VENTRICLES IN DIASTOLE SHOULD BE THE SAME AMOUNG THAT GOES
OUT)3-ELEVATED LVDP.4-MR SCONDARY 2 TO LV AND ANULAR
DILATION.5-PULMONARY HYPERTENTION.6-LV DIASTOLE DYSFUNTION.OR LV NON
COMPLIANCE...SYMPTOMS??..EXERSICE INTOLERANCE..APNEA(SHORTENESS OF
BREATH)..FINDING ECHOCARDIOGRAPHY??..(INCREASED SPHERICAL OF THE LV
GEOMETRY..APICAL AND LATERAL DISPLACEMENT OF THE PAPILLARY MUSCLES DUE
TO THE ANNULAR DILATATION...LV THROMBUS DUE TO THE POOL STASIS OF
BLOOD AND POOR CO)...LV ENLARGEMENT LEADS TO AND AF(ATRIAL
FIBRILATION,COMMON ARRHYTHMIA REFERED AS PATIENT WITH ELECTRICAL OR
CHEMICAL CARDIOVERTION)AF DU TO A PROLONGED DILATATION.LOST OF ATRIAL
KICK OR SYSTOLE..CONSECUENSES??REDUSED CO(CARDIAC OUTPUT AND
CONGESTIVE HEART FAILURE.....M-MODE FINDING??..DECREASE STROKE VOLUME
RESULT IN LESS ONDULATION OF THE AORTA..DECREASED AORTIC
EXCURTION..FLAT ROOT MOTION..LA,RV ENLARGEMENT..GRADUAL CLOSURE OF THE
AORTA DURING SYSTOLE..MINIMAL OPENNING OF THE LEAFLETS..REDUCE
ANTEROPOSTRIOR AO ROOY MOTION...::)))))

very nice clip