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A ligated Left Atrial Appendage
Prior to cardioversion, this patient with prior CABG supposedly had the LAA ligated. The indentation in the atrium is the remnant of the LAA. Notice the faint spontaneous echo \smoke\.
Runtime: 0m:1s
Prior to cardioversion, this patient with prior CABG supposedly had the LAA ligated. The indentation in the atrium is the remnant of the LAA. Notice the faint spontaneous echo \smoke\.
Runtime: 0m:1s
Adult with congenital heart defect (Part 2 of 2)
Patient with septic shock and congenital heart defect: in TEE we could see an ASD, a VSD and we had an idea, that there is a sinus venosus defect and a parietal thrombus in the right pulmonary artery too.
Runtime: 0m:40s
Patient with septic shock and congenital heart defect: in TEE we could see an ASD, a VSD and we had an idea, that there is a sinus venosus defect and a parietal thrombus in the right pulmonary artery too.
Runtime: 0m:40s
Aneurysm of the atrial septum with PFO
Aneurysm of the atrial septum with PFO documented with echovist in TEE
Runtime: 0m:34s
Aneurysm of the atrial septum with PFO documented with echovist in TEE
Runtime: 0m:34s
Another HOCM Part 2
here the TEE of further patient with severe HOCM with SAM of AMS and following high grade mitral valve insufficiency and fluttering aortic valve respectively mesosystolic closure of aortiv valve (unfortunately I still can´t convert my M-Mode-pictures of SAM and mesosystolic closure of aortic valve).
Runtime: 0m:30s
here the TEE of further patient with severe HOCM with SAM of AMS and following high grade mitral valve insufficiency and fluttering aortic valve respectively mesosystolic closure of aortiv valve (unfortunately I still can´t convert my M-Mode-pictures of SAM and mesosystolic closure of aortic valve).
Runtime: 0m:30s
aortic regurgitation due to aortic cusp prolapse
severe excentric aortic regurgitation due to significant anterior aortic cusp prolapse.TEE.
Runtime: 0m:48s
severe excentric aortic regurgitation due to significant anterior aortic cusp prolapse.TEE.
Runtime: 0m:48s
Aortic valve vegetation 1 of 2
In this TEE clip of an asymptomatic 80 year old man, this vegetation was discovered incidentally.
Runtime: 0m:2s
In this TEE clip of an asymptomatic 80 year old man, this vegetation was discovered incidentally.
Runtime: 0m:2s
Aortic valve vegetation 2 of 2
This TEE clip demonstrates a vegetation on the aortic valve which was found incidentally in an asymptomatic 80 year old man. This clip was taken with a biplane/3D probe and two simultaneous views.
Runtime: 0m:2s
This TEE clip demonstrates a vegetation on the aortic valve which was found incidentally in an asymptomatic 80 year old man. This clip was taken with a biplane/3D probe and two simultaneous views.
Runtime: 0m:2s
biventricular failure caused by septic shock
about 80 y old patient with septic shock caused by severe pulmonary infection (gram-stain: gram-negative rod cells > pseudomonas). TEE showed a biventricular hypodynamic septic cardiomyopathy, beside we could see a mitral insuffiziency (2. grade; PISA 0,6), no reflux in pulmonary veins. We administered high dose catecholamines (norepinephrin, dobutamin), so we could stabilize the haemodynamic clinical situation.
Runtime: 0m:27s
about 80 y old patient with septic shock caused by severe pulmonary infection (gram-stain: gram-negative rod cells > pseudomonas). TEE showed a biventricular hypodynamic septic cardiomyopathy, beside we could see a mitral insuffiziency (2. grade; PISA 0,6), no reflux in pulmonary veins. We administered high dose catecholamines (norepinephrin, dobutamin), so we could stabilize the haemodynamic clinical situation.
Runtime: 0m:27s
cardiogenic shock caused by posterolateral infarction (IABP)
This clip shows you the echocardiographic picture of posterolateral infarction after implantation of IABP. After 1 day of implantation of IABP PA-cath showed following results: SVR 820 dyn*s*cm^-5, cardiac output 3,8 l/min, cardiac power 0,9 W!
Runtime: 0m:34s
This clip shows you the echocardiographic picture of posterolateral infarction after implantation of IABP. After 1 day of implantation of IABP PA-cath showed following results: SVR 820 dyn*s*cm^-5, cardiac output 3,8 l/min, cardiac power 0,9 W!
Runtime: 0m:34s
cardiogenic shock caused by posterolateral-infarction (p-IABP implantation)
about 85 y old patient with STEMI. cardiac catheter showed an occlusion of ramus circumflecus. In TTE and TEE we could see an akinesia of posterolateral wall. In PA-cath: SVR 1200 dyn*s*cm^-5, CO 2,8 l/min, cardiac power 0,51 W: this is compatible to the clinical and echocardiographic picture of cardiogenic shock! This clip shows cardiac wall movemnt disorder and restricted cardiac ejection fraction prae-implantation of IABP.
Runtime: 0m:17s
about 85 y old patient with STEMI. cardiac catheter showed an occlusion of ramus circumflecus. In TTE and TEE we could see an akinesia of posterolateral wall. In PA-cath: SVR 1200 dyn*s*cm^-5, CO 2,8 l/min, cardiac power 0,51 W: this is compatible to the clinical and echocardiographic picture of cardiogenic shock! This clip shows cardiac wall movemnt disorder and restricted cardiac ejection fraction prae-implantation of IABP.
Runtime: 0m:17s
cardiogenic shock caused by thrombembolic occlusion of left main stem
about 75 y old patient with STEMI and about 45 minutes cpr. the patient was admitted from our ER after cardiac catheter to our ICU. In cardiac catheter we found a thrombotic occlusion of left main stem, no plaques and no stenosis. in clinical history a permanent atrial fibrillation with condition after several thrombembolic strokes is known. in TEE we found a distinct cardiac wall movement disorder of left ventricle and a small parietal residual of thrombus in LAA. we think that a thrombembolic cardiac infarction caused by thrombus in LAA is the most plausible reason of this finding.
Runtime: 0m:19s
about 75 y old patient with STEMI and about 45 minutes cpr. the patient was admitted from our ER after cardiac catheter to our ICU. In cardiac catheter we found a thrombotic occlusion of left main stem, no plaques and no stenosis. in clinical history a permanent atrial fibrillation with condition after several thrombembolic strokes is known. in TEE we found a distinct cardiac wall movement disorder of left ventricle and a small parietal residual of thrombus in LAA. we think that a thrombembolic cardiac infarction caused by thrombus in LAA is the most plausible reason of this finding.
Runtime: 0m:19s
chordal rupture of both mitral leaflets:infective endocarditis
infective endocarditis:chordal rupture of both mitral leaflets with vegetations on both leaflets and severe mitral regurgitation.TEE.
Runtime: 0m:35s
infective endocarditis:chordal rupture of both mitral leaflets with vegetations on both leaflets and severe mitral regurgitation.TEE.
Runtime: 0m:35s
chordal rupture of the posterior mitral leaflet with flail mitral valve
infective endocarditis:vegetation and chordal rupture of the posterior mitral leaflet with flail mitral valve(and severe mitral regurgitation).poor echogenicity on TTE and better study on TEE (done just before surgery).
Runtime: 0m:44s
infective endocarditis:vegetation and chordal rupture of the posterior mitral leaflet with flail mitral valve(and severe mitral regurgitation).poor echogenicity on TTE and better study on TEE (done just before surgery).
Runtime: 0m:44s
chordal rupture with severe mitral regurgitation TEE 1/2
TEE shows the ruptured chordae ( anterior leaflet) moving between the LV during diastole and the LA during systole
Runtime: 0m:8s
TEE shows the ruptured chordae ( anterior leaflet) moving between the LV during diastole and the LA during systole
Runtime: 0m:8s
chordal rupture with severe mitral regurgitation TEE 2/2
TEE and color doppler recording shows the severe mitral regurgitation
Runtime: 0m:14s
TEE and color doppler recording shows the severe mitral regurgitation
Runtime: 0m:14s
Condition after cardiac surgery
about 35 y old patient with condition after mitral-valve reconstruction (Carpentier-ring 36 mm) because of severe high-grad symptomatic mitral-valve insufficiency caused by mitral-valve prolapse and implantation of mechanical aortic prosthesis because of bicuspid aortic valve (SJM double tilting disk-prosthesis 25 mm). You can see a small prolapse of posterior mitral valve but no relevant m itral valve insufficiency, no elevated transprothetic gradient (PGmean 2,7 mmHg). I used tissue-imagening to demonstrate mitral-valve movement.
Runtime: 0m:47s
about 35 y old patient with condition after mitral-valve reconstruction (Carpentier-ring 36 mm) because of severe high-grad symptomatic mitral-valve insufficiency caused by mitral-valve prolapse and implantation of mechanical aortic prosthesis because of bicuspid aortic valve (SJM double tilting disk-prosthesis 25 mm). You can see a small prolapse of posterior mitral valve but no relevant m itral valve insufficiency, no elevated transprothetic gradient (PGmean 2,7 mmHg). I used tissue-imagening to demonstrate mitral-valve movement.
Runtime: 0m:47s
Cor pulmonale chronicum
About 70 y old patient with chronic dyspnoea, permanent atrial fibrillation (CHADS-Vasc-Scor 5), actually deterioration of clinical afflictions. A COPD is kown (stage III GOLD) In TTE and TEE we ccould see a distinct RV-hypertrophy with systolic and diastolic septal bounce (D-sign) as a sign for RV-volume overload and RV-pressure overload (PAPsystolic about 70 mmHg + CVP). In LAA you can see a distinct echocantrast (thrombus?). An another interesting finding is a PFO with right-to-left-shunt.
Runtime: 1m:20s
About 70 y old patient with chronic dyspnoea, permanent atrial fibrillation (CHADS-Vasc-Scor 5), actually deterioration of clinical afflictions. A COPD is kown (stage III GOLD) In TTE and TEE we ccould see a distinct RV-hypertrophy with systolic and diastolic septal bounce (D-sign) as a sign for RV-volume overload and RV-pressure overload (PAPsystolic about 70 mmHg + CVP). In LAA you can see a distinct echocantrast (thrombus?). An another interesting finding is a PFO with right-to-left-shunt.
Runtime: 1m:20s
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